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Dr. Ali explains Chelation and natural remedies for
Reversing Heart Disease Part 1
Reversing Heart Disease Part 2
Preventing first and subsequent heart attacks
Bundle 2 $19.95
This is a four video set
running approximately 3 hours combined. In it Dr. Ali discuss
the following topics:
Atrial Fibrillation Seminar
Heart Valve Diseases Part 1
Heart Valve Diseases Part 2
Heart - Diseased
or Overdriven? Seminar 1 - Course on Heart Health
In this 45-minute Part 1 of “Heart - Diseased or Overdriven?
Seminar of “Dr. Ali’s Course on Heart Health,” Professor Majid
Ali addresses the grave error of confusing an overdriven heart
with a diseased heart. Hundreds of million people worldwide live
in fear of being a “heart patient” when all they need is to
learn what an overdriven heart is and how to calm it. This
concerns people with heart palpitations, skipped heart beats,
tachycardia due to excess of adrenaline, acidity, free radicals,
insulin, and cortisone. It also concerns people who have had
coronary stents or bypasses which were really not necessary. The
matter of medical emergencies is different. Prof. Ali offers
information and expects viewers to decide if he is overstating
Heart - Diseased
or Overdriven? Seminar 2 - Course on Heart Health
In this 40-minute Part 2 of "Heart - Sick or Overdriven? Seminar
of "Dr. Ali’s Course on Heart Health," Professor Majid Ali
further underscores the grave error of confusing an overdriven
heart with a diseased heart. Hundreds of million people
worldwide live in fear of being a "heart patient" when all they
need is to learn what an overdriven heart is and how to calm it.
Acute medical emergencies, he advises, require treatment by
experienced doctors with excess to necessary technology. He then
guidelines for natural non-drug remedies to "calm the heart" and
avoid the perils which an overdriven heart faces if the
overdrive state persist. Prof. Ali offers information and
expects viewers to decide if he is overstating his case.
In this 50-minute video, Professor Majid Ali, M.D. presents oral
and intravenous chelation therapies for reversing heart disease,
as well as for removing mercury, lead other tosic metals from
the body. Prof. Ali draws upon his five decades of experience in
surgery, pathology, immunology, nutritional medicine, and
chelation therapies to offer clinically proven chelation
Part 1 In
this 40-minute video seminar, Professor Majid Ali, M.D.
describes his integrative plan to reverse heart disease. He
emphasizes the need for consulting experienced doctors for acute
heart conditions. Medications for heart disease must not be
discontinued until non-drug remedies become effective and drugs
can be discontinued safely under a doctor’s care. In his
unifying Oxygen Model of Heart Disease, he looks at the disease
holistically and integratively.
Part 2 In
this 50-minute Part 2 of a video seminar on reversing heart
disease, Professor Majid Ali, M.D. presents in detail his
integrative plan for the reversal of heart disease with focus on
spices, herbs, anti-inflammatory oil rubs, and natural
oxygen-enhancing remedies. He emphasizes the need not stopping
heart medications until nondrug remedies work, especially Limbic
Breathing to slow the heart rate and normalize blood pressure.
Palipitations of parents and grand parents
Heart palpitations are a common and distressing problem for a
large number of senior citizens. Their children and
grandchildren can do much with natural remedies to prevent and
control palpitations. In this 40-minute video seminar, Professor
Majid Ali, M.D. discusses the causes, clinical features, types,
and nondrug control of heart palpitations.
In this 45-minute video seminar, Professor Majid Ali explains
why coronary stents do not save lives except in true
emergencies. He cites published met-analysis of large number of
stent studies in support of his statement and offers guidelines
for natural remedies for heart health.
Diseases Seminar 1 -Dr. Ali’s Course on Heart Health
In this 30-minute video seminar, Professor Majid Ali, M.D.
presents the causes, signs and symptoms, clinical course, and
consequences of diseases of heart valve, including
calcifications and stenosis. For many individuals, the diagnosis
of these conditions have no serious clinical significance.
Mitral valve prolapse is an excellent example.
valves Seminar 2 - Course on Heart Health
In this 35-minute part 2
video seminar, Professor Majid Ali, M.D. continues his
presentation of the causes, signs and symptoms, clinical course,
and consequences of heart valve calcifications and diseases.
Saying No HEART
In the prevailing drug model, all initial triggers, which can be
blocked with simple natural remedies, are ignored. What is left
then are drugs, unnecessary coronary bypass operations, coronary
stents, and radio-destruction of heart pacemakers.
In this 40-minute video seminar, Professor Majid Ali, M.D.
describes the causes, clinical features, and non-drug treatment
of atrial fibrillation (A-Fib). It is the most common form of
heart rhythm disturbances (up to 9% of people over 75% are
affected). It is caused by inflammation and scarring that
damaged specialized cells that make up the heart pacemaker. This
chamber is often stretched, which further stresses the
More arterial plaques are associated with fewer
deaths from heart attacks and strokes. No, you did not misread
the sentence. I write precisely what I mean: people with less
intense plaque formation have higher mortality from
cardiovascular diseases. I anticipate harsh frowns from doctors
on the title of this tutorial. The idea that plaques in arteries
cause heart attacks and strokes is deeply etched in doctors’
belief systems, and through them in the consciousness of the
general public. The very notion that more plaques are associated
with fewer heart attacks and strokes is likely to be considered
Strong Claims Require Strong
Neglect of the scientific facts of arterial
plaques leads to more disastrous treatment errors than any other
neglect except that of insulin toxicity that sets the stage for
obesity and diabetes. This is a strong claims and requires
strong evidence. I devote this five-part series to do just that.
I present personal autopsy, microscopic, biochemical, and
clinical observations to expose the folly of both the plaque
theory and cholesterol theories of coronary heart disease. I
also cite the neglected published work of others to further
support my assertions.
In past publications, I presented extensive
evidence for debunking the "cholesterol theory" of coronary
heart disease. In this series, I marshal evidence to debunk the
"plaque theory"—the notion that plaques cause heart attacks—to
offer readers a liberating and scientifically sound challenge to
the dictums of the bypass-stent industry for cardiac conditions
that do not threaten life in crisis setting.
I introduce the term "more-plaques-fewer-deaths
paradox" to highlight six essential points.
First, in general fewer coronary heart
and stroke deaths are associated with greater degrees of
arterial plaques. Surprise!
have choice: We can keep the inner "endo region" of the
arterial wall healthy and live longer, or we can obsess
on cholesterol deposits and plaques in the outer region
of the arterial wall and live less. Another surprise!
countless lives are lost when non-drug oxygen therapies
to restore "endo health" are opposed and blocker drugs
(beta blockers, calcium channel bloclers, etc) are used
to suppress symptoms caused by injury to endo cells.
coronary bypasses and stents do not remove arterial
plaques. Stents close up with high frequency and
bypasses less often.
people live long healthful lives after coronary bypasses
and stents but only when they address issues of foods,
environment, an dstress that keep the inner linings of
arteries non-sticky and negatively charged.
save lives in crisis situations but do not reverse
plaque formation to any meaningful degrees. By contrast,
the focus on the inner endo region, where the
circulating blood interfaces with the arterial wall,
prevents heart attacks, strokes, and deaths.
An Iron Pipe With Inner Aluminum
Imagine an iron water pipe with a delicate inner
aluminum lining. The lining is so delicate that it is easily
injured and leaks water. The lining also has a magical capacity
for self-healing. It readily plugs its own micro-holes and
prevents further water leakage. However it cannot prevent slow
and steady corrosion of the outer strong part of the pipe made
of iron by water which leaks through it before it self-plugs.
One part of the iron-aluminum pipe is burried in
the soil. With passing years, the corroded iron part of the wall
develops microscopic holes and some water leaking through tears
in the aluminum lining,escapes into the soil surrounding the
pipe. With time, the leaked water feeds mold spores in the soil
and other microbes to create moldy, mulch-like suds around the
pipe. A CAT scan of the pipe at this time reveals a thickened
pipe wall—muddy mulch suds appear like plaques. However, the
technology of CAT scanning is not yet advanced to reveal
self-healing tears in the inner delicate aluminum lining. When
the water flowing through the pipe is checked, it is clear.
Evolution’s Intelligent Design
The structure of arteries is one of Nature’s
marvel. It is a self-cleansing and self-healing structure. My
grandfather forgot to die on time. I do not know whether he
lived for 101, 102, or more years. My grandmother lived a
similarly long life. They did not have any heart attacks or
strokes. The Japanese in Okinawa Islands live for 120 or more
years without heart attacks or strokes. That means Nature’s
intelligent design for arteries can prevent plaque formation for
hundred or more years, if only toxicities of foods, environment,
and thoughts can be avoided.
Anatomy of the Arterial Wall
The arterial wall has the following four layers:
1. Inner endothelial (endo for short) lining
composed of a single-celled layer
2. A thin layer of loose connective tissue
beneath the endo cells.
3. A strong and resilient muscle layer
4. A thin outermost layer of loose connective
The inner endo layer is smooth and has a negative
charge. It forms the "inner region" and prevents the blood cells
and plasma from sticking to it. The outer region is where muscle
and surrounding tissue develop cholesterol crystals, calcium
deposits, and plaques. The endo region directly faces the
circulating blood while the outer region is removed from it. The
endo cells have strong healing ability since they are directly
supplied by oxygen and nutrients. By contrast, the outer part
damaged by scarring and plaques have limited ability to heal and
restore their normal structure.
The "action" in events that trigger heart
attacks, strokes, and kidney failure occurs in the endo
region—the "endo-blood interface" seems an appropriate
designation—and is always regulated and preserved by factors
related to oxygen homeostasis (balance). This simple fact is
readily validated by high-resolution phase-contrast microscopy.
Regrettably, it is completely neglected by doctors controlled by
the "heart disease industrial mega-complex." Please ask
yourself, when did I last see an "endo commercial" on TV? Or,
when did The New York Times publish a large "endo article"? Or,
The New Enron Journal of Medicine fight back the "statin
monsters" who want everyone to be on drugs like Lipitor, Crestor,
Zocor, Pravaco an dothers prescribed to lower blood cholesterol
What Do Arterial Plaques Really
In cardiology literature, those who profit from
the plaque preoccupation seldom, if ever, duly recognize and
consider evidence against their model of the plaque-death
relationships. Below are some quotes from recent cardiology
journals that reveal the dimension of the problem that are
seldom, if ever, duly considered in the matters of plaque-death
1. Atherosclerotic plaques that lead to acute
coronary syndromes often occur at sites of angiographically mild
coronary-artery stenosis. Lesion-related risk factors for such
events are poorly understood (N Engl J Med 2011; 364:226-235).
2. Currently, there are no available methods that
can reliably predict when or if an atheroma will rupture. (
Journal of Invasive Cardiology 2010;22: 406-411, September
3. The study by Stone et al. provides conclusive
evidence that current morphologic assessments of vulnerable
coronary plaque do not predict major adverse cardiovascular
4. Study4 have confirmed that positive remodeling
was a predictor of future acute coronary events, independent of
5. In response to the comments of Maseri and
colleagues (writers of a letter to the Editor) we agree that the
lesion-specific characteristics that we identified were
sensitive but not specific for future major adverse
6. The 13 infarctions (areas of tissue death)
that were related to culprit lesions were caused by stent
Robust Endo Responses Explain the
The leaking iron-aluminum pipe analogy explains
the absence of the cholesterol-coronary connection, but not the
more-plaques-fewer deaths paradox. For this, one must understand
diverse tissue healing responses under varying conditions. Acute
injury triggers rapid but short-durated response. Chronic
ongoing tissue insult, in contrast, evoke persistent healing
responses. Such differences are well known to pathologists. Up
to a point, the more repeated the insult, the stronger the
healing response. Plaque formation in arteries occurs as a
delayed consequence of repeated injury to the endo cells, which
would be expected to mount stronger and longer lasting "endo-healing"
responses. That, indeed, occurs. For example, a compensatory
enlargement of human coronary arteries occurs in some cases of
advanced plaque formation.
True Significance of Arterial
Are plaques in the arteries of the heart, brain,
and kidneys of no significance? No. Plaques indicate the
presence of chronic vascular injury. The longer the duration of
vascular injury, the more wide spread the plaques. The crucial
clinical importance of more-plaques-fewer-deaths paradox is: It
fundamentally challenges the prevailing view of the cause of
heart attacks and strokes. It shifts the focus from secondary
changes in the outer arterial wall—plaques, cholesterol
crystals, and calcium deposits—to the crucial primary
events involving the
inner endo cells. Heart attacks are triggered when endo cells in
the coronary arteries are injured beyond their ability to repair
themselves. Strokes are caused when the same happens to endo
cells lining brain arteries. And so begins the kidney damage
that often leads to dialysis.
In my Oxygen Model of Coronary Artery Disease,
micro-clots and micro-plaques in the circulating blood form and
endo dysfunction develops due to local dysox conditions
(deranged oxygen signals and other aspects of dysfunctional
oxygen metabolism). Dysox sets the stage for excess acidity,
increased oxyradical activity, and thickening of bodily fluids.
Simply stated, heart attacks and strokes are the problems of the
"oxygen-endo (ox-en) axis"—the centerpiece of the Oxygen Model.